GipA Factor Supports Colonization of Peyerʼs Patches by Crohnʼs Disease-associated Escherichia Coli (VSports手机版)

VSports app下载. Nine of the 41 strains harboured the afaC gene (H504, H305, H020, H296, 61–1 ti1, 52–2 ti10, 52–1 ti3, 18–4 ti12, 18–3 ti5), 4 of which were AIEC (36% of all AIEC strains), belonged to ST95 and had an O18:H7 serotype. …”Section: Resultsmentioningconfidence: 99%

"V体育2025版" Comparative genomics of Crohn's disease-associated adherent-invasiveEscherichia coli

O’Brien1, Bringer2, Holt3 et al. 2016GutSelf Cite104111090Get access via publisherAdd to dashboardCite Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task. Show abstract Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task. “…We conducted presence/absence and phylogenetic analyses on the following LF82-associated genes: rpoE ;39 htrA (also known as degP and ptd );40 dsbA, 41 hfq, 42 and AIEC/IBD-associated E. coli genes : pduC 29 (propanediol utilisation), fyuA, ibeA, K1, kpsmTII, 17 vgrG , hcp , vasD , vasG , impL , impK (type VI secretion system genes), and vat , insA , insB, 30 fimH, 28 afaC 45 and clb (pks island),46and the combination of gipA and lpfA, 43 or lpfA alone 44 45. Nine of the 41 strains harboured the afaC gene (H504, H305, H020, H296, 61–1 ti1, 52–2 ti10, 52–1 ti3, 18–4 ti12, 18–3 ti5), 4 of which were AIEC (36% of all AIEC strains), belonged to ST95 and had an O18:H7 serotype. …”Section: Resultsmentioningconfidence: 99%

Comparative genomics of Crohn's disease-associated adherent-invasiveEscherichia coli

O’Brien1, Bringer2, Holt3 et al. 2016GutSelf Cite104111090Get access via publisherAdd to dashboardCite Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task. Show abstract Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task. “…GipA expression is induced by certain intracellular, GI (bile salts) and phagolysosomal (ROS and acid pH) conditions. Deletion of gipA dampens the proinflammatory response and AIEC replication inside macrophages 53. AIEC killing by macrophages could be inhibited by microbial mannan in a TLR4 and MyD88-dependent manner 132…”Section: Aiec and The Gut Immune Systemmentioningconfidence: 99%

Adherent-invasive Escherichia coli in inflammatory bowel disease

Palmela1, Chevarin2, Xu3 et al. 2017GutSelf Cite470133740Get access via publisherAdd to dashboardCite Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task. Show abstract Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task. show abstract“…[137][138][139] Adherence and invasion of pathobionts usually requires expression of specific genes such as proteases in E. faecalis and GipA (growth in Peyer' s patches) in AIEC. [140][141][142][143] In addition to attachment and invasion, pathobiont species also impair normal IEC functions for intestinal homeostasis. C. concisus, a gram-negative microbe that is increased in pediatric and adult IBD patients, inhibits expression of the tight junction-associated proteins ZO-1 and occludin, as well as their association with tight junction complexes leading to a less regulated intestinal barrier. …”Section: Alterations Of Intestinal Epithelial Integritymentioningconfidence: 99%

"V体育安卓版" Epithelial-microbial diplomacy: escalating border tensions drive inflammation in inflammatory bowel disease

King1, McCole2 2019Intest Res172100Get access via publisherAdd to dashboardCite Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task. Show abstract Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task. show abstractScite is an AI-powered research tool that helps researchers better discover and evaluate scientific literature through Smart Citations—a revolutionary system that shows whether articles support, contrast, or simply mention a given claim. Founded in 2018, and now part of Research Solutions, Scite has indexed over 1. 3 billion citations and partnered with more than 30 major publishers to provide researchers with unparalleled access to scientific literature. With its Scite Assistant, Smart Citation Index, and advanced search capabilities, the platform addresses critical challenges such as information overload and research reproducibility. Trusted by two million active users worldwide, Scite is reshaping how researchers interact with scholarly content—building ethical, transparent AI tools that support rigorous, copyright-compliant research.